Publicaciones en las que colabora con MARCOS FERNANDEZ FONDEVILA (22)

2024

  1. Inhibition of hepatic p63 ameliorates steatohepatitis with fibrosis in mice

    Molecular Metabolism, Vol. 85

  2. The lipopolysaccharide-TLR4 axis regulates hepatic glutaminase 1 expression promoting liver ammonia build-up as steatotic liver disease progresses to steatohepatitis

    Metabolism: Clinical and Experimental, Vol. 158

  3. p63 controls metabolic activation of hepatic stellate cells and fibrosis via an HER2-ACC1 pathway

    Cell Reports Medicine, Vol. 5, Núm. 2

2023

  1. Hepatic levels of S-adenosylmethionine regulate the adaptive response to fasting

    Cell Metabolism, Vol. 35, Núm. 8, pp. 1373-1389.e8

  2. Hepatic p63 regulates glucose metabolism by repressing SIRT1

    Gut, Vol. 72, Núm. 3, pp. 472-483

  3. Hepatocyte-specific O-GlcNAc transferase downregulation ameliorates nonalcoholic steatohepatitis by improving mitochondrial function

    Molecular Metabolism, Vol. 75

  4. Neddylation of phosphoenolpyruvate carboxykinase 1 controls glucose metabolism

    Cell Metabolism, Vol. 35, Núm. 9, pp. 1630-1645.e5

2022

  1. Inhibition of ATG3 ameliorates liver steatosis by increasing mitochondrial function

    Journal of Hepatology, Vol. 76, Núm. 1, pp. 11-24

  2. Inhibition of carnitine palmitoyltransferase 1A in hepatic stellate cells protects against fibrosis

    Journal of Hepatology, Vol. 77, Núm. 1, pp. 15-28

  3. O-GlcNAcylation: A Sweet Hub in the Regulation of Glucose Metabolism in Health and Disease

    Frontiers in Endocrinology, Vol. 13

2021

  1. Metabolic Landscape of the Mouse Liver by Quantitative 31P Nuclear Magnetic Resonance Analysis of the Phosphorome

    Hepatology, Vol. 74, Núm. 1, pp. 148-163

  2. O-GlcNAcylated p53 in the liver modulates hepatic glucose production

    Nature Communications, Vol. 12, Núm. 1

  3. Obese patients with NASH have increased hepatic expression of SARS-CoV-2 critical entry points

    Journal of Hepatology

  4. Papel del sistema L-α-lisofosfatidilinositol/GPR55 en el desarrollo de esteatosis no alcohólica y esteatohepatitis

    Papel del sistema L-α-lisofosfatidilinositol/GPR55 en el desarrollo de esteatosis no alcohólica y esteatohepatitis

  5. Splicing factor SF3B1 is overexpressed and implicated in the aggressiveness and survival of hepatocellular carcinoma

    Cancer Letters, Vol. 496, pp. 72-83

  6. The L-α-Lysophosphatidylinositol/G Protein–Coupled Receptor 55 System Induces the Development of Nonalcoholic Steatosis and Steatohepatitis

    Hepatology, Vol. 73, Núm. 2, pp. 606-624

2019

  1. Hypothalamic dopamine signalling regulates brown fat thermogenesis

    Nature Metabolism, Vol. 1, Núm. 8, pp. 811-829

2018

  1. Hypothalamic GRP78, a new target against obesity?

    Adipocyte

  2. Pharmacological stimulation of p53 with low-dose doxorubicin ameliorates diet-induced nonalcoholic steatosis and steatohepatitis

    Molecular Metabolism, Vol. 8, pp. 132-143

  3. p53 in AgRP neurons is required for protection against diet-induced obesity via JNK1

    Nature Communications, Vol. 9, Núm. 1