Publicacións nas que colabora con RAMON SOTO OTERO (17)

2024

  1. Copper Overload Increased Rat Striatal Levels of Both Dopamine and Its Main Metabolite Homovanillic Acid in Extracellular Fluid

    International Journal of Molecular Sciences, Vol. 25, Núm. 15

2019

  1. Copper Increases Brain Oxidative Stress and Enhances the Ability of 6-Hydroxydopamine to Cause Dopaminergic Degeneration in a Rat Model of Parkinson’s Disease

    Molecular Neurobiology, Vol. 56, Núm. 4, pp. 2845-2854

2017

  1. Effects of Aluminium on Rat Brain Mitochondria Bioenergetics: an In vitro and In vivo Study

    Molecular Neurobiology, Vol. 54, Núm. 1, pp. 563-570

2016

  1. Mitochondrial angiotensin receptors in dopaminergic neurons. Role in cell protection and aging-related vulnerability to neurodegeneration

    Cell Death and Disease, Vol. 7, Núm. 10

2013

  1. A simple method for isolating rat brain mitochondria with high metabolic activity: Effects of EDTA and EGTA

    Journal of Neuroscience Methods, Vol. 213, Núm. 1, pp. 39-42

2012

  1. 2-Benzazepine nitrones protect dopaminergic neurons against 6-hydroxydopamine-induced oxidative toxicity

    Archiv der Pharmazie, Vol. 345, Núm. 8, pp. 598-609

2009

  1. Brain oxidative stress and selective behaviour of aluminium in specific areas of rat brain: Potential effects in a 6-OHDA-induced model of Parkinson's disease

    Journal of Neurochemistry, Vol. 109, Núm. 3, pp. 879-888

2007

  1. Angiotensin type-1-receptor antagonists reduce 6-hydroxydopamine toxicity for dopaminergic neurons

    Neurobiology of Aging, Vol. 28, Núm. 4, pp. 555-567

  2. Time-course of brain oxidative damage caused by intrastriatal administration of 6-hydroxydopamine in a rat model of Parkinson's disease

    Neurochemical Research, Vol. 32, Núm. 1, pp. 99-105

2006

  1. Reduction of dopaminergic degeneration and oxidative stress by inhibition of angiotensin converting enzyme in a MPTP model of parkinsonism

    Neuropharmacology, Vol. 51, Núm. 1, pp. 112-120

2005

  1. Angiotensin-converting enzyme inhibition reduces oxidative stress and protects dopaminergic neurons in a 6-hydroxydopamine rat model of Parkinsonism

    Journal of Neuroscience Research, Vol. 81, Núm. 6, pp. 865-873

2004

  1. Systemic Administration of N-Acetylcysteine Protects Dopaminergic Neurons Against 6-Hydroxydopamine-Induced Degeneration

    Journal of Neuroscience Research, Vol. 76, Núm. 4, pp. 551-562

2002

  1. Effects of (-)-nicotine and (-)-cotinine on 6-hydroxydopamine-induced oxidative stress and neurotoxicity: Relevance for Parkinson's disease

    Biochemical Pharmacology, Vol. 64, Núm. 1, pp. 125-135

  2. Effects of aluminum and zinc on the oxidative stress caused by 6-hydroxydopamine autoxidation: Relevance for the pathogenesis of Parkinson's disease

    Biochimica et Biophysica Acta - Molecular Basis of Disease, Vol. 1586, Núm. 2, pp. 155-168

2001

  1. Effect of iron and manganese on hydroxyl radical production by 6-hydroxydopamine: Mediation of antioxidants

    Free Radical Biology and Medicine, Vol. 31, Núm. 8, pp. 986-998

  2. N-acetylcysteine enhances production of dopaminergic neurons from mesencephalic-derived precursor cells

    NeuroReport, Vol. 12, Núm. 18, pp. 3935-3938

2000

  1. Autoxidation and neurotoxicity of 6-hydroxydopamine in the presence of some antioxidants: Potential implication in relation to the pathogenesis of Parkinson's disease

    Journal of Neurochemistry, Vol. 74, Núm. 4, pp. 1605-1612