Department of Morphological Science
Department
RAMON
SOTO OTERO
Ad honorem Emeritus professor
Publications by the researcher in collaboration with RAMON SOTO OTERO (17)
2024
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Copper Overload Increased Rat Striatal Levels of Both Dopamine and Its Main Metabolite Homovanillic Acid in Extracellular Fluid
International Journal of Molecular Sciences, Vol. 25, Núm. 15
2019
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Copper Increases Brain Oxidative Stress and Enhances the Ability of 6-Hydroxydopamine to Cause Dopaminergic Degeneration in a Rat Model of Parkinson’s Disease
Molecular Neurobiology, Vol. 56, Núm. 4, pp. 2845-2854
2017
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Effects of Aluminium on Rat Brain Mitochondria Bioenergetics: an In vitro and In vivo Study
Molecular Neurobiology, Vol. 54, Núm. 1, pp. 563-570
2016
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Mitochondrial angiotensin receptors in dopaminergic neurons. Role in cell protection and aging-related vulnerability to neurodegeneration
Cell Death and Disease, Vol. 7, Núm. 10
2013
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A simple method for isolating rat brain mitochondria with high metabolic activity: Effects of EDTA and EGTA
Journal of Neuroscience Methods, Vol. 213, Núm. 1, pp. 39-42
2012
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2-Benzazepine nitrones protect dopaminergic neurons against 6-hydroxydopamine-induced oxidative toxicity
Archiv der Pharmazie, Vol. 345, Núm. 8, pp. 598-609
2009
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Brain oxidative stress and selective behaviour of aluminium in specific areas of rat brain: Potential effects in a 6-OHDA-induced model of Parkinson's disease
Journal of Neurochemistry, Vol. 109, Núm. 3, pp. 879-888
2007
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Angiotensin type-1-receptor antagonists reduce 6-hydroxydopamine toxicity for dopaminergic neurons
Neurobiology of Aging, Vol. 28, Núm. 4, pp. 555-567
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Time-course of brain oxidative damage caused by intrastriatal administration of 6-hydroxydopamine in a rat model of Parkinson's disease
Neurochemical Research, Vol. 32, Núm. 1, pp. 99-105
2006
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Reduction of dopaminergic degeneration and oxidative stress by inhibition of angiotensin converting enzyme in a MPTP model of parkinsonism
Neuropharmacology, Vol. 51, Núm. 1, pp. 112-120
2005
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Angiotensin-converting enzyme inhibition reduces oxidative stress and protects dopaminergic neurons in a 6-hydroxydopamine rat model of Parkinsonism
Journal of Neuroscience Research, Vol. 81, Núm. 6, pp. 865-873
2004
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Systemic Administration of N-Acetylcysteine Protects Dopaminergic Neurons Against 6-Hydroxydopamine-Induced Degeneration
Journal of Neuroscience Research, Vol. 76, Núm. 4, pp. 551-562
2002
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Effects of (-)-nicotine and (-)-cotinine on 6-hydroxydopamine-induced oxidative stress and neurotoxicity: Relevance for Parkinson's disease
Biochemical Pharmacology, Vol. 64, Núm. 1, pp. 125-135
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Effects of aluminum and zinc on the oxidative stress caused by 6-hydroxydopamine autoxidation: Relevance for the pathogenesis of Parkinson's disease
Biochimica et Biophysica Acta - Molecular Basis of Disease, Vol. 1586, Núm. 2, pp. 155-168
2001
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Effect of iron and manganese on hydroxyl radical production by 6-hydroxydopamine: Mediation of antioxidants
Free Radical Biology and Medicine, Vol. 31, Núm. 8, pp. 986-998
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N-acetylcysteine enhances production of dopaminergic neurons from mesencephalic-derived precursor cells
NeuroReport, Vol. 12, Núm. 18, pp. 3935-3938
2000
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Autoxidation and neurotoxicity of 6-hydroxydopamine in the presence of some antioxidants: Potential implication in relation to the pathogenesis of Parkinson's disease
Journal of Neurochemistry, Vol. 74, Núm. 4, pp. 1605-1612