Publicacións en colaboración con investigadores/as de Centro de Investigaciones Energéticas, Medioambientales y Tecnológicas (34)

2022

  1. Development of a mouse model for spontaneous oral squamous cell carcinoma in Fanconi anemia

    Oral Oncology, Vol. 134

2021

  1. Eras, a member of the ras superfamily, acts as an oncoprotein in the mammary gland

    Cancers, Vol. 13, Núm. 21

2020

  1. IKKβ overexpression together with a lack of tumour suppressor genes causes ameloblastic odontomas in mice

    International Journal of Oral Science, Vol. 12, Núm. 1

2019

  1. Premature aging and cancer development in transgenic mice lacking functional CYLD

    Aging, Vol. 11, Núm. 1, pp. 127-159

2017

  1. A transposon-based analysis reveals RASA1 is involved in triple-negative breast cancer

    Cancer Research, Vol. 77, Núm. 6, pp. 1357-1368

  2. Context-dependent role of IKKβ in cancer

    Genes, Vol. 8, Núm. 12

  3. IKKβ-mediated resistance to skin cancer development is Ink4a/Arf-dependent

    Molecular Cancer Research, Vol. 15, Núm. 9, pp. 1255-1264

2016

  1. IKKα regulates the stratification and differentiation of the epidermis: Implications for skin cancer development

    Oncotarget, Vol. 7, Núm. 47, pp. 76779-76792

  2. Protective role of p53 in skin cancer: Carcinogenesis studies in mice lacking epidermal p53

    Oncotarget, Vol. 7, Núm. 15, pp. 20902-20918

2014

  1. P21 suppresses inflammation and tumorigenesis on pRB-deficient stratified epithelia

    Oncogene, Vol. 33, Núm. 37, pp. 4599-4612

  2. RNAi-mediated knockdown of IKK1 in transgenic mice using a transgenic construct containing the human H1 promoter

    The Scientific World Journal, Vol. 2014

2013

  1. A transposon-based analysis of gene mutations related to skin cancer development

    Journal of Investigative Dermatology, Vol. 133, Núm. 1, pp. 239-248

2011

  1. CYLD regulates keratinocyte differentiation and skin cancer progression in humans

    Cell Death and Disease, Vol. 2, Núm. 9

  2. IKKβ overexpression leads to pathologic lesions in stratified epithelia and exocrine glands and to tumoral transformation of oral epithelia

    Molecular Cancer Research, Vol. 9, Núm. 10, pp. 1329-1338

  3. Increased IKKα expression in the basal layer of the epidermis of transgenic mice enhances the malignant potential of skin tumors

    PLoS ONE, Vol. 6, Núm. 7

2010

  1. An inactivating CYLD mutation promotes skin tumor progression by conferring enhanced proliferative, survival and angiogenic properties to epidermal cancer cells

    Oncogene, Vol. 29, Núm. 50, pp. 6522-6532

  2. IKKΒ leads to an inflammatory skin disease resembling interface dermatitis

    Journal of Investigative Dermatology, Vol. 130, Núm. 6, pp. 1598-1610

2009

  1. Akt activation synergizes with Trp53 loss in oral epithelium to produce a novel mouse model for head and neck squamous cell carcinoma

    Cancer Research, Vol. 69, Núm. 3, pp. 1099-1108

2008

  1. Constitutively active Akt induces ectodermal defects and impaired bone morphogenetic protein signaling

    Molecular Biology of the Cell, Vol. 19, Núm. 1, pp. 137-149

  2. IKKα enhances human keratinocyte differentiation and determines the histological variant of epidermal squamous cell carcinomas

    Cell Cycle, Vol. 7, Núm. 13, pp. 2021-2029