Publicacións nas que colabora con José Luis Cascallana (11)

2023

  1. Modification of the Marmarou and Foda model of diffuse axonal injury (DAI) improves percentage survival of rats at 24 h and increases the amount of DAI identified

    Journal of Forensic Sciences, Vol. 68, Núm. 4, pp. 1228-1236

2011

  1. IKKβ overexpression leads to pathologic lesions in stratified epithelia and exocrine glands and to tumoral transformation of oral epithelia

    Molecular Cancer Research, Vol. 9, Núm. 10, pp. 1329-1338

2010

  1. IKKΒ leads to an inflammatory skin disease resembling interface dermatitis

    Journal of Investigative Dermatology, Vol. 130, Núm. 6, pp. 1598-1610

2008

  1. Constitutively active Akt induces ectodermal defects and impaired bone morphogenetic protein signaling

    Molecular Biology of the Cell, Vol. 19, Núm. 1, pp. 137-149

  2. Glucocorticoid receptor is required for skin barrier competence

    Endocrinology, Vol. 149, Núm. 3, pp. 1377-1388

  3. Transrepression function of the glucocorticoid receptor regulates eyelid development and keratinocyte proliferation but is not sufficient to prevent skin chronic inflammation

    Molecular Endocrinology, Vol. 22, Núm. 4, pp. 799-812

2007

  1. Deregulated activity of Akt in epithelial basal cells induces spontaneous tumors and heightened sensitivity to skin carcinogenesis

    Cancer Research, Vol. 67, Núm. 22, pp. 10879-10888

2005

  1. Ectoderm-targeted overexpression of the glucocorticoid receptor induces hypohidrotic ectodermal dysplasia

    Endocrinology, Vol. 146, Núm. 6, pp. 2629-2638

  2. Estudio fenotípico de ratones transgénicos con sobreexpresión del receptor de glucocorticoides

    Estudio fenotípico de ratones transgénicos con sobreexpresión del receptor de glucocorticoides

  3. Skin immune responses in cattle after primary and secondary infections with Hypoderma lineatum (Diptera: Oestridae) larvae

    Veterinary Immunology and Immunopathology, Vol. 108, Núm. 3-4, pp. 285-294

2003

  1. Disruption of eyelid and cornea development by targeted overexpression of the glucocorticoid receptor

    International Journal of Developmental Biology, Vol. 47, Núm. 1, pp. 59-64