Publikationen in Zusammenarbeit mit Forschern von Massachusetts General Hospital (8)

2008

  1. SOK1 translocates from the golgi to the nucleus upon chemical anoxia and induces apoptotic cell death

    Journal of Biological Chemistry, Vol. 283, Núm. 23, pp. 16248-16258

1997

  1. Activation of the Ste20-like oxidant stress response kinase-1 during the initial stages of chemical anoxia-induced necrotic cell death. Requirement for dual inputs of oxidant stress and increased cytosolic [Ca2+]

    Journal of Biological Chemistry, Vol. 272, Núm. 46, pp. 29372-29379

1996

  1. Activation of a human Ste20-like kinase by oxidant stress defines a novel stress response pathway

    EMBO Journal, Vol. 15, Núm. 17, pp. 4537-4546

  2. Partial characterization of a putative new growth factor present in pathological human vitreous

    Graefe's Archive for Clinical and Experimental Ophthalmology, Vol. 234, Núm. 3, pp. 155-163

  3. Stress-activated protein kinases in cardiovascular disease

    Circulation Research

1995

  1. Activation of the SAPK pathway by the human STE20 homologue germinal centre kinase

    Nature, Vol. 377, Núm. 6551, pp. 750-754

  2. Ischemia and reperfusion enhance ATF-2 and c-Jun binding to cAMP response elements and to an AP-1 binding site from the c-jun promoter

    Journal of Biological Chemistry, Vol. 270, Núm. 50, pp. 30084-30092

1994

  1. The stress-activated protein kinases are major c-Jun amino-terminal kinases activated by ischemia and reperfusion

    Journal of Biological Chemistry, Vol. 269, Núm. 42, pp. 26546-26551