Publications in collaboration with researchers from University of Cambridge (36)

2022

  1. Kappa-Opioid Receptor Blockade Ameliorates Obesity Caused by Estrogen Withdrawal via Promotion of Energy Expenditure through mTOR Pathway

    International Journal of Molecular Sciences, Vol. 23, Núm. 6

2021

  1. Sirt3 in POMC neurons controls energy balance in a sex- and diet-dependent manner

    Redox Biology, Vol. 41

2020

  1. Brain-Sparing Sympathofacilitators Mitigate Obesity without Adverse Cardiovascular Effects

    Cell Metabolism, Vol. 31, Núm. 6, pp. 1120-1135.e7

2019

  1. Hypothalamic dopamine signalling regulates brown fat thermogenesis

    Nature Metabolism, Vol. 1, Núm. 8, pp. 811-829

  2. MCH regulates SIRT1/FoxO1 and reduces POMC neuronal activity to induce hyperphagia, adiposity, and glucose intolerance

    Diabetes, Vol. 68, Núm. 12, pp. 2210-2222

  3. Thyroid-Hormone-Induced Browning of White Adipose Tissue Does Not Contribute to Thermogenesis and Glucose Consumption

    Cell Reports, Vol. 27, Núm. 11, pp. 3385-3400.e3

2018

  1. Melanin-Concentrating Hormone acts through hypothalamic kappa opioid system and p70S6K to stimulate acute food intake

    Neuropharmacology, Vol. 130, pp. 62-70

  2. p53 in AgRP neurons is required for protection against diet-induced obesity via JNK1

    Nature Communications, Vol. 9, Núm. 1

2017

  1. A brain-sparing diphtheria toxin for chemical genetic ablation of peripheral cell lineages

    Nature Communications, Vol. 8

  2. Hypothalamic AMPK-ER Stress-JNK1 Axis Mediates the Central Actions of Thyroid Hormones on Energy Balance

    Cell Metabolism, Vol. 26, Núm. 1, pp. 212-229.e12

2014

  1. The central nervous system in metabolic syndrome

    A Systems Biology Approach to Study Metabolic Syndrome (Springer International Publishing), pp. 137-156

2013

  1. Adaptive changes of the Insig1/SREBP1/SCD1 set point help adipose tissue to cope with increased storage demands of obesity

    Diabetes, Vol. 62, Núm. 11, pp. 3697-3708

2012

  1. Acute effects of orexigenic antipsychotic drugs on lipid and carbohydrate metabolism in rat

    Psychopharmacology, Vol. 219, Núm. 3, pp. 783-794

  2. BMP8B increases brown adipose tissue thermogenesis through both central and peripheral actions

    Cell, Vol. 149, Núm. 4, pp. 871-885

  3. Hypothalamic mTOR pathway mediates thyroid hormone-induced hyperphagia in hyperthyroidism

    Journal of Pathology, Vol. 227, Núm. 2, pp. 209-222

  4. Nicotine induces negative energy balance through hypothalamic AMP-activated protein kinase

    Diabetes, Vol. 61, Núm. 4, pp. 807-817

  5. Olanzapine, but not aripiprazole, weight-independently elevates serum triglycerides and activates lipogenic gene expression in female rats

    International Journal of Neuropsychopharmacology, Vol. 15, Núm. 2, pp. 163-179

2011

  1. Ghrelin and lipid metabolism: Key partners in energy balance

    Journal of Molecular Endocrinology, Vol. 46, Núm. 2, pp. 43-63

  2. Hypothalamic AMP-activated protein kinase as a mediator of whole body energy balance

    Reviews in Endocrine and Metabolic Disorders, Vol. 12, Núm. 3, pp. 127-140

  3. Olanzapine-induced hyperphagia and weight gain associate with orexigenic hypothalamic neuropeptide signaling without concomitant AMPK phosphorylation

    PLoS ONE, Vol. 6, Núm. 6