DIANA
GUALLAR ARTAL
Investigadora contratada Ramón y Cajal
![Foto de DIANA](/img/nophoto.png)
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Icahn School of Medicine at Mount Sinai
Nueva York, Estados UnidosPublicacións en colaboración con investigadores/as de Icahn School of Medicine at Mount Sinai (13)
2020
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ADAR1-Dependent RNA Editing Promotes MET and iPSC Reprogramming by Alleviating ER Stress
Cell Stem Cell, Vol. 27, Núm. 2, pp. 300-314.e11
2018
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RNA-dependent chromatin targeting of TET2 for endogenous retrovirus control in pluripotent stem cells
Nature Genetics, Vol. 50, Núm. 3, pp. 443-451
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YY1 Positively Regulates Transcription by Targeting Promoters and Super-Enhancers through the BAF Complex in Embryonic Stem Cells
Stem Cell Reports, Vol. 10, Núm. 4, pp. 1324-1339
2017
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Context-Dependent Functions of NANOG Phosphorylation in Pluripotency and Reprogramming
Stem Cell Reports, Vol. 8, Núm. 5, pp. 1115-1123
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NAC1 Regulates Somatic Cell Reprogramming by Controlling Zeb1 and E-cadherin Expression
Stem Cell Reports, Vol. 9, Núm. 3, pp. 913-926
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The SIN3A/HDAC Corepressor Complex Functionally Cooperates with NANOG to Promote Pluripotency
Cell Reports, Vol. 18, Núm. 7, pp. 1713-1726
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Zfp281 is essential for mouse epiblast maturation through transcriptional and epigenetic control of Nodal signaling
eLife, Vol. 6
2016
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Taking the RISC of exiting naïve pluripotency
Genome biology, Vol. 17, pp. 104
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Zfp281 Coordinates Opposing Functions of Tet1 and Tet2 in Pluripotent States
Cell Stem Cell, Vol. 19, Núm. 3, pp. 355-369
2015
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Tex10 Coordinates Epigenetic Control of Super-Enhancer Activity in Pluripotency and Reprogramming
Cell Stem Cell, Vol. 16, Núm. 6, pp. 653-668
2014
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A genome-wide RNAi screen identifies opposing functions of snai1 and snai2 on the nanog dependency in reprogramming
Molecular Cell, Vol. 56, Núm. 1, pp. 140-152
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RNA-binding proteins in pluripotency, differentiation, and reprogramming
Frontiers in Biology, Vol. 9, Núm. 5, pp. 389-409
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Regulation of mouse retroelement MuERV-L/MERVL expression by REX1 and epigenetic control of stem cell potency
Frontiers in Oncology, Vol. 4 FEB