Publicacións en colaboración con investigadores/as de Icahn School of Medicine at Mount Sinai (13)

2020

  1. ADAR1-Dependent RNA Editing Promotes MET and iPSC Reprogramming by Alleviating ER Stress

    Cell Stem Cell, Vol. 27, Núm. 2, pp. 300-314.e11

2018

  1. RNA-dependent chromatin targeting of TET2 for endogenous retrovirus control in pluripotent stem cells

    Nature Genetics, Vol. 50, Núm. 3, pp. 443-451

  2. YY1 Positively Regulates Transcription by Targeting Promoters and Super-Enhancers through the BAF Complex in Embryonic Stem Cells

    Stem Cell Reports, Vol. 10, Núm. 4, pp. 1324-1339

2017

  1. Context-Dependent Functions of NANOG Phosphorylation in Pluripotency and Reprogramming

    Stem Cell Reports, Vol. 8, Núm. 5, pp. 1115-1123

  2. NAC1 Regulates Somatic Cell Reprogramming by Controlling Zeb1 and E-cadherin Expression

    Stem Cell Reports, Vol. 9, Núm. 3, pp. 913-926

  3. The SIN3A/HDAC Corepressor Complex Functionally Cooperates with NANOG to Promote Pluripotency

    Cell Reports, Vol. 18, Núm. 7, pp. 1713-1726

  4. Zfp281 is essential for mouse epiblast maturation through transcriptional and epigenetic control of Nodal signaling

    eLife, Vol. 6

2016

  1. Taking the RISC of exiting naïve pluripotency

    Genome biology, Vol. 17, pp. 104

  2. Zfp281 Coordinates Opposing Functions of Tet1 and Tet2 in Pluripotent States

    Cell Stem Cell, Vol. 19, Núm. 3, pp. 355-369

2015

  1. Tex10 Coordinates Epigenetic Control of Super-Enhancer Activity in Pluripotency and Reprogramming

    Cell Stem Cell, Vol. 16, Núm. 6, pp. 653-668

2014

  1. A genome-wide RNAi screen identifies opposing functions of snai1 and snai2 on the nanog dependency in reprogramming

    Molecular Cell, Vol. 56, Núm. 1, pp. 140-152

  2. RNA-binding proteins in pluripotency, differentiation, and reprogramming

    Frontiers in Biology, Vol. 9, Núm. 5, pp. 389-409

  3. Regulation of mouse retroelement MuERV-L/MERVL expression by REX1 and epigenetic control of stem cell potency

    Frontiers in Oncology, Vol. 4 FEB