Mecanismos moleculares da enfermedade
MeMoEn
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Massachusetts General Hospital
Boston, Estados UnidosPublicacións en colaboración con investigadores/as de Massachusetts General Hospital (9)
2020
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Pan-cancer analysis of whole genomes identifies driver rearrangements promoted by LINE-1 retrotransposition
Nature Genetics, Vol. 52, Núm. 3, pp. 306-319
2008
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SOK1 translocates from the golgi to the nucleus upon chemical anoxia and induces apoptotic cell death
Journal of Biological Chemistry, Vol. 283, Núm. 23, pp. 16248-16258
1997
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Activation of the Ste20-like oxidant stress response kinase-1 during the initial stages of chemical anoxia-induced necrotic cell death. Requirement for dual inputs of oxidant stress and increased cytosolic [Ca2+]
Journal of Biological Chemistry, Vol. 272, Núm. 46, pp. 29372-29379
1996
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Activation of a human Ste20-like kinase by oxidant stress defines a novel stress response pathway
EMBO Journal, Vol. 15, Núm. 17, pp. 4537-4546
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Partial characterization of a putative new growth factor present in pathological human vitreous
Graefe's Archive for Clinical and Experimental Ophthalmology, Vol. 234, Núm. 3, pp. 155-163
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Stress-activated protein kinases in cardiovascular disease
Circulation Research
1995
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Activation of the SAPK pathway by the human STE20 homologue germinal centre kinase
Nature, Vol. 377, Núm. 6551, pp. 750-754
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Ischemia and reperfusion enhance ATF-2 and c-Jun binding to cAMP response elements and to an AP-1 binding site from the c-jun promoter
Journal of Biological Chemistry, Vol. 270, Núm. 50, pp. 30084-30092
1994
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The stress-activated protein kinases are major c-Jun amino-terminal kinases activated by ischemia and reperfusion
Journal of Biological Chemistry, Vol. 269, Núm. 42, pp. 26546-26551