CALHM1 and its polymorphism P86L differentially control Ca2+ homeostasis, mitogen-activated protein kinase signaling, and cell vulnerability upon exposure to amyloid β

1. Moreno-Ortega, A.J.
2. Buendia, I.
3. Mouhid, L.
4. Egea, J.
5. Lucea, S.
6. Ruiz-Nuño, A.
7. López, M.G.
8. Cano-Abad, M.F.

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DOI: 10.1111/ACEL.12403 GOOGLE SCHOLAR lock_openOpen access editor

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